Schizophrenia and fragile X syndrome both can involve problems with cognitive function, and two studies published in Nature Neuroscience earlier on August 4 identify a gene that may underlie the neurodevelopmental problems associated with the diseases.
The gene, topoisomerase 3β (TOP3β), encodes an enzyme that interacts with FMRP, a protein that causes fragile X syndrome when mutated, and the researchers suspect that the proteins’ interactions promote normal cognitive development.
Read the full, original story here: Gene Spans Mental Disorders